mouse cxcl2 gro beta mip Search Results


94
Bio-Techne corporation mouse cxcl2/gro beta/mip-2/cinc-3 biotinylated antibody
Mouse Cxcl2/Gro Beta/Mip 2/Cinc 3 Biotinylated Antibody, supplied by Bio-Techne corporation, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 94 stars, based on 1 article reviews
mouse cxcl2/gro beta/mip-2/cinc-3 biotinylated antibody - by Bioz Stars, 2026-09
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Proteintech cxcl2
Chemokine profile of the control and LPS-induced Raw264.7 EVs. (A) The chemokine profile of EVs from control and LPS-induced Raw264.7 cells. (B) The <t>CXCL2</t> level (mean ± SD) in the control-Raw-EV and LPS-Raw-EV groups. The experiments were repeated three times. **p < 0.01 versus the control-Raw264.7-EV group. (C) PKH67-labeled control-Raw264.7-EVs and LPS-Raw264.7-EVs were injected into wild-type C57BL/6 mice (n = 6 per group) via the tail vein. The colocalization of PKH67-EVs and CXCL2 in the liver was detected by immunofluorescence (original magnification × 200). (D and E) GO analysis and KEGG analysis of various chemokines expressed in EVs from control and LPS-induced Raw264.7 cells.
Cxcl2, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mouse+cxcl2+gro+beta+mip/Mouse+CXCL2+ELISA+Kit/pmc08490939-219-3-5
Average 93 stars, based on 1 article reviews
cxcl2 - by Bioz Stars, 2026-09
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94
R&D Systems anti cxcl2
Chemokine profile of the control and LPS-induced Raw264.7 EVs. (A) The chemokine profile of EVs from control and LPS-induced Raw264.7 cells. (B) The <t>CXCL2</t> level (mean ± SD) in the control-Raw-EV and LPS-Raw-EV groups. The experiments were repeated three times. **p < 0.01 versus the control-Raw264.7-EV group. (C) PKH67-labeled control-Raw264.7-EVs and LPS-Raw264.7-EVs were injected into wild-type C57BL/6 mice (n = 6 per group) via the tail vein. The colocalization of PKH67-EVs and CXCL2 in the liver was detected by immunofluorescence (original magnification × 200). (D and E) GO analysis and KEGG analysis of various chemokines expressed in EVs from control and LPS-induced Raw264.7 cells.
Anti Cxcl2, supplied by R&D Systems, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mouse+cxcl2+gro+beta+mip/Mouse+CXCL2%2FGRO+beta%2FMIP-2%2FCINC-3+Antibody/pmc11319398-41-20-23
Average 94 stars, based on 1 article reviews
anti cxcl2 - by Bioz Stars, 2026-09
94/100 stars
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R&D Systems anti mip 2 antibody
Chemokine profile of the control and LPS-induced Raw264.7 EVs. (A) The chemokine profile of EVs from control and LPS-induced Raw264.7 cells. (B) The <t>CXCL2</t> level (mean ± SD) in the control-Raw-EV and LPS-Raw-EV groups. The experiments were repeated three times. **p < 0.01 versus the control-Raw264.7-EV group. (C) PKH67-labeled control-Raw264.7-EVs and LPS-Raw264.7-EVs were injected into wild-type C57BL/6 mice (n = 6 per group) via the tail vein. The colocalization of PKH67-EVs and CXCL2 in the liver was detected by immunofluorescence (original magnification × 200). (D and E) GO analysis and KEGG analysis of various chemokines expressed in EVs from control and LPS-induced Raw264.7 cells.
Anti Mip 2 Antibody, supplied by R&D Systems, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mouse+cxcl2+gro+beta+mip/Mouse+CXCL2%2FGRO+beta%2FMIP-2%2FCINC-3+Antibody/10__1165_slash_rcmb__2003___0373oc-77-23-31
Average 94 stars, based on 1 article reviews
anti mip 2 antibody - by Bioz Stars, 2026-09
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R&D Systems mouse cxcl2 gro beta mip
Chemokine profile of the control and LPS-induced Raw264.7 EVs. (A) The chemokine profile of EVs from control and LPS-induced Raw264.7 cells. (B) The <t>CXCL2</t> level (mean ± SD) in the control-Raw-EV and LPS-Raw-EV groups. The experiments were repeated three times. **p < 0.01 versus the control-Raw264.7-EV group. (C) PKH67-labeled control-Raw264.7-EVs and LPS-Raw264.7-EVs were injected into wild-type C57BL/6 mice (n = 6 per group) via the tail vein. The colocalization of PKH67-EVs and CXCL2 in the liver was detected by immunofluorescence (original magnification × 200). (D and E) GO analysis and KEGG analysis of various chemokines expressed in EVs from control and LPS-induced Raw264.7 cells.
Mouse Cxcl2 Gro Beta Mip, supplied by R&D Systems, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mouse+cxcl2+gro+beta+mip/Recombinant+Mouse+CXCL2%2FGRO+beta%2FMIP-2%2FCINC-3+Protein/pm38091995-766-322-326
Average 94 stars, based on 1 article reviews
mouse cxcl2 gro beta mip - by Bioz Stars, 2026-09
94/100 stars
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90
ImmunoTools recombinant mouse growth factor beta (rm gro-beta / rm cxcl2 / rm mip-2a)
Chemokine profile of the control and LPS-induced Raw264.7 EVs. (A) The chemokine profile of EVs from control and LPS-induced Raw264.7 cells. (B) The <t>CXCL2</t> level (mean ± SD) in the control-Raw-EV and LPS-Raw-EV groups. The experiments were repeated three times. **p < 0.01 versus the control-Raw264.7-EV group. (C) PKH67-labeled control-Raw264.7-EVs and LPS-Raw264.7-EVs were injected into wild-type C57BL/6 mice (n = 6 per group) via the tail vein. The colocalization of PKH67-EVs and CXCL2 in the liver was detected by immunofluorescence (original magnification × 200). (D and E) GO analysis and KEGG analysis of various chemokines expressed in EVs from control and LPS-induced Raw264.7 cells.
Recombinant Mouse Growth Factor Beta (Rm Gro Beta / Rm Cxcl2 / Rm Mip 2a), supplied by ImmunoTools, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mouse+cxcl2+gro+beta+mip/recombinant+mouse+growth+factor+beta++rm+gro+beta+++rm+cxcl2+++rm+mip+2a+/pm39120532-79-5-3
Average 90 stars, based on 1 article reviews
recombinant mouse growth factor beta (rm gro-beta / rm cxcl2 / rm mip-2a) - by Bioz Stars, 2026-09
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N/A
The Mouse CXCL2 GRO beta MIP 2 CINC 3 Antibody from R D Systems is a rat monoclonal antibody to CXCL2 GRO beta MIP 2 CINC 3 This antibody reacts with mouse The Mouse CXCL2
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N/A
Recombinant Mouse CXCL2/GRO beta/MIP-2/CINC-3 Protein
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mGRO b; Recombinant Mouse GRO-beta/MIP-2 (CXCL2); Recombinant Mouse GRO-beta/MIP-2 (CXCL2)
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N/A
The Recombinant Mouse CXCL2 GRO beta MIP 2 CINC 3 Protein from Novus Biologicals is derived from E coli The Recombinant Mouse CXCL2 GRO beta MIP 2 CINC 3 Protein has been validated for the
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N/A
Simple Plex Control for Mouse CXCL2/GRO beta/MIP-2/CINC-3
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Image Search Results


Chemokine profile of the control and LPS-induced Raw264.7 EVs. (A) The chemokine profile of EVs from control and LPS-induced Raw264.7 cells. (B) The CXCL2 level (mean ± SD) in the control-Raw-EV and LPS-Raw-EV groups. The experiments were repeated three times. **p < 0.01 versus the control-Raw264.7-EV group. (C) PKH67-labeled control-Raw264.7-EVs and LPS-Raw264.7-EVs were injected into wild-type C57BL/6 mice (n = 6 per group) via the tail vein. The colocalization of PKH67-EVs and CXCL2 in the liver was detected by immunofluorescence (original magnification × 200). (D and E) GO analysis and KEGG analysis of various chemokines expressed in EVs from control and LPS-induced Raw264.7 cells.

Journal: The Journal of Immunology Author Choice

Article Title: Macrophagic Extracellular Vesicle CXCL2 Recruits and Activates the Neutrophil CXCR2/PKC/NOX4 Axis in Sepsis

doi: 10.4049/jimmunol.2100229

Figure Lengend Snippet: Chemokine profile of the control and LPS-induced Raw264.7 EVs. (A) The chemokine profile of EVs from control and LPS-induced Raw264.7 cells. (B) The CXCL2 level (mean ± SD) in the control-Raw-EV and LPS-Raw-EV groups. The experiments were repeated three times. **p < 0.01 versus the control-Raw264.7-EV group. (C) PKH67-labeled control-Raw264.7-EVs and LPS-Raw264.7-EVs were injected into wild-type C57BL/6 mice (n = 6 per group) via the tail vein. The colocalization of PKH67-EVs and CXCL2 in the liver was detected by immunofluorescence (original magnification × 200). (D and E) GO analysis and KEGG analysis of various chemokines expressed in EVs from control and LPS-induced Raw264.7 cells.

Article Snippet: The quantification of CXCL2 (KE10022; Proteintech), CXCL7 (DY393; R&D Systems, Minneapolis, MN), CXCL17 (AD7670Hu; Andy Gene, Beijing, China), CCL2 (KE00091; Proteintech), CCL3 (KE00092; Proteintech), and CCL5 (KE00093; Proteintech) was performed using quantitative ELISA.

Techniques: Control, Labeling, Injection, Immunofluorescence

LPS-induced CXCL2 of Raw264.7 EVs attracts neutrophils in vitro and in vivo and activates neutrophils via the CXCR2/PKC/NOX4 pathway. (A) The CXCL2 level (mean ± SD) of Raw264.7 in the control, LPS, LPS + lenti-CXCL2, and LPS + siRNA-CXCL2 groups detected by Western blotting (WB). The experiments were repeated three times. *p < 0.01 versus the control group, &p < 0.01 versus the LPS group, #p < 0.01 versus the LPS group. (B) The CXCL2 level (mean ± SD) in Raw264.7 EVs of the control, LPS, LPS + lenti-CXCL2, and LPS + siRNA-CXCL2 groups detected by WB. The experiments were repeated three times. (C) Raw264.7 EVs derived from the control, LPS, LPS + lenti-CXCL2, and LPS + siRNA-CXCL2 groups induced BMDN chemotaxis (mean ± SD) (n = 5 fields per group). (D) The expression of CXCR2, PKC, and NOX4 (mean ± SD) of BMDNs in the control EVs, LPS-EVs, (LPS + lenti-CXCL2)-EVs, and (LPS + siRNA-CXCL2)-EVs groups was detected by WB. The experiments were repeated three times. (E) H&E staining, MPO+ cells, and Ly-6G+ cells (mean ± SD) in the liver of the control EVs, LPS-EVs, (LPS + lenti-CXCL2)-EVs, and (LPS + siRNA-CXCL2)-EV group mice (n = 6 per group) detected by immunohistochemistry. Scale bars, 100 μm. (F–H) ALT, AST, and LDH levels (mean ± SD) in the serum of the control EVs, LPS-EVs, (LPS + lenti-CXCL2)-EVs, and (LPS + siRNA-CXCL2)-EV group mice (n = 6 per group). (I) The number of systemic circulating neutrophils (mean ± SD) in wild-type C57BL/6 mice treated with control EVs, LPS-EVs, (LPS + lenti-CXCL2)-EVs, and (LPS + siRNA-CXCL2)-EVs (n = 3 per group). (J) The expression of CXCR2, PKC, and NOX4 (mean ± SD) in the liver of the control EVs, LPS-EVs, (LPS + lenti-CXCL2)-EVs, and (LPS + siRNA-CXCL2)-EV group mice was detected by WB. The experiments were repeated three times. *p < 0.01 versus the control-Raw264.7-EV group, &p < 0.01 versus the LPS-Raw264.7-EV group, #p < 0.01 versus the LPS-Raw264.7-EV group.

Journal: The Journal of Immunology Author Choice

Article Title: Macrophagic Extracellular Vesicle CXCL2 Recruits and Activates the Neutrophil CXCR2/PKC/NOX4 Axis in Sepsis

doi: 10.4049/jimmunol.2100229

Figure Lengend Snippet: LPS-induced CXCL2 of Raw264.7 EVs attracts neutrophils in vitro and in vivo and activates neutrophils via the CXCR2/PKC/NOX4 pathway. (A) The CXCL2 level (mean ± SD) of Raw264.7 in the control, LPS, LPS + lenti-CXCL2, and LPS + siRNA-CXCL2 groups detected by Western blotting (WB). The experiments were repeated three times. *p < 0.01 versus the control group, &p < 0.01 versus the LPS group, #p < 0.01 versus the LPS group. (B) The CXCL2 level (mean ± SD) in Raw264.7 EVs of the control, LPS, LPS + lenti-CXCL2, and LPS + siRNA-CXCL2 groups detected by WB. The experiments were repeated three times. (C) Raw264.7 EVs derived from the control, LPS, LPS + lenti-CXCL2, and LPS + siRNA-CXCL2 groups induced BMDN chemotaxis (mean ± SD) (n = 5 fields per group). (D) The expression of CXCR2, PKC, and NOX4 (mean ± SD) of BMDNs in the control EVs, LPS-EVs, (LPS + lenti-CXCL2)-EVs, and (LPS + siRNA-CXCL2)-EVs groups was detected by WB. The experiments were repeated three times. (E) H&E staining, MPO+ cells, and Ly-6G+ cells (mean ± SD) in the liver of the control EVs, LPS-EVs, (LPS + lenti-CXCL2)-EVs, and (LPS + siRNA-CXCL2)-EV group mice (n = 6 per group) detected by immunohistochemistry. Scale bars, 100 μm. (F–H) ALT, AST, and LDH levels (mean ± SD) in the serum of the control EVs, LPS-EVs, (LPS + lenti-CXCL2)-EVs, and (LPS + siRNA-CXCL2)-EV group mice (n = 6 per group). (I) The number of systemic circulating neutrophils (mean ± SD) in wild-type C57BL/6 mice treated with control EVs, LPS-EVs, (LPS + lenti-CXCL2)-EVs, and (LPS + siRNA-CXCL2)-EVs (n = 3 per group). (J) The expression of CXCR2, PKC, and NOX4 (mean ± SD) in the liver of the control EVs, LPS-EVs, (LPS + lenti-CXCL2)-EVs, and (LPS + siRNA-CXCL2)-EV group mice was detected by WB. The experiments were repeated three times. *p < 0.01 versus the control-Raw264.7-EV group, &p < 0.01 versus the LPS-Raw264.7-EV group, #p < 0.01 versus the LPS-Raw264.7-EV group.

Article Snippet: The quantification of CXCL2 (KE10022; Proteintech), CXCL7 (DY393; R&D Systems, Minneapolis, MN), CXCL17 (AD7670Hu; Andy Gene, Beijing, China), CCL2 (KE00091; Proteintech), CCL3 (KE00092; Proteintech), and CCL5 (KE00093; Proteintech) was performed using quantitative ELISA.

Techniques: In Vitro, In Vivo, Control, Western Blot, Derivative Assay, Chemotaxis Assay, Expressing, Staining, Immunohistochemistry

EVs from the serum of patients with sepsis attract neutrophils and activate the neutrophil CXCR2/PKC/NOX4 pathway in vitro. (A) The chemokine profile of EVs from the serum of healthy individuals and patients with sepsis. (B–G) CXCL2, CXCL7, CXCL17, CCL2, CCL3, and CCL5 levels (mean ± SD) in EVs from the serum of healthy individuals (n = 8) and patients with sepsis (n = 26) detected using ELISA. *p < 0.05 versus the healthy-EV group, ***p < 0.01 versus the healthy-EV group. (H) sEVs derived from healthy control (HC) and patients with sepsis-induced PMN chemotaxis (mean ± SD) (n = 5 fields per group). (I) The expression of CXCR2, PKC, and NOX4 (mean ± SD) in the PMNs of the HC-sEV– and sepsis-sEV–treated groups was detected by Western blotting. The experiments were repeated three times. **p < 0.01 versus the healthy-EV group, #p < 0.05 versus the healthy-EV group, &p < 0.05 versus the healthy-EV group.

Journal: The Journal of Immunology Author Choice

Article Title: Macrophagic Extracellular Vesicle CXCL2 Recruits and Activates the Neutrophil CXCR2/PKC/NOX4 Axis in Sepsis

doi: 10.4049/jimmunol.2100229

Figure Lengend Snippet: EVs from the serum of patients with sepsis attract neutrophils and activate the neutrophil CXCR2/PKC/NOX4 pathway in vitro. (A) The chemokine profile of EVs from the serum of healthy individuals and patients with sepsis. (B–G) CXCL2, CXCL7, CXCL17, CCL2, CCL3, and CCL5 levels (mean ± SD) in EVs from the serum of healthy individuals (n = 8) and patients with sepsis (n = 26) detected using ELISA. *p < 0.05 versus the healthy-EV group, ***p < 0.01 versus the healthy-EV group. (H) sEVs derived from healthy control (HC) and patients with sepsis-induced PMN chemotaxis (mean ± SD) (n = 5 fields per group). (I) The expression of CXCR2, PKC, and NOX4 (mean ± SD) in the PMNs of the HC-sEV– and sepsis-sEV–treated groups was detected by Western blotting. The experiments were repeated three times. **p < 0.01 versus the healthy-EV group, #p < 0.05 versus the healthy-EV group, &p < 0.05 versus the healthy-EV group.

Article Snippet: The quantification of CXCL2 (KE10022; Proteintech), CXCL7 (DY393; R&D Systems, Minneapolis, MN), CXCL17 (AD7670Hu; Andy Gene, Beijing, China), CCL2 (KE00091; Proteintech), CCL3 (KE00092; Proteintech), and CCL5 (KE00093; Proteintech) was performed using quantitative ELISA.

Techniques: In Vitro, Enzyme-linked Immunosorbent Assay, Derivative Assay, Control, Chemotaxis Assay, Expressing, Western Blot